![]() Results: For acute COVID-19 patients, group mean (range) for PA-aO2 was 41.4 (-3.5-69.3) mmHg and for Pa-ACO2 was 6.0 (-2.3-13.4) mmHg. Arterial blood (breathing ambient air) was collected while exhaled oxygen and carbon dioxide concentrations were measured, yielding alveolar-arterial differences for each gas (PA-aO2 and Pa-ACO2, respectively) from which shunt and alveolar dead space were computed. Methods: We studied 30 patients (22 males mean +/- SD age 49.9 +/- 13.5 years) 3-15 days from symptom onset and again during recovery, 55 +/- 10 days later (n=17). We aimed to measure shunt and alveolar dead space in moderate COVID-19 during acute illness and recovery. We propose that measuring respiratory gas exchange enables detection and quantification of these abnormalities. 61, no 1, article id 2201117 Article in journal (Refereed) Published Abstract īackground: Pathological evidence suggests that coronavirus disease 2019 (COVID-19) pulmonary infection involves both alveolar damage (causing shunt) and diffuse microvascular thrombus formation (causing alveolar dead space). Show others and affiliations 2023 (English) In: European Respiratory Journal, ISSN 0903-1936, E-ISSN 1399-3003, Vol.
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